M-CSF-induced VEGF production from human monocytes is blocked by ERK inhibition and mithramycin.    From Wordnik.com.  [PLoS ONE Alerts: New Articles] Reference

Next, we isolated RNA from cells pre-incubated with U1026, mithramycin, or vehicle controls and treated with M-CSF.    From Wordnik.com.  [PLoS ONE Alerts: New Articles] Reference

It has been reported earlier that thiazolidinediones (TZDs), could not repress the expression of resistin gene in the presence of mithramycin A (Sp1 binding inhibitor) in 3T3-L1 adipocytes.    From Wordnik.com.  [PLoS ONE Alerts: New Articles] Reference

(NS) or stimulated with rhM-CSF (100 ng / ml) (M-CSF), (M-CSF+MeOH), (M-CSF plus 8 nM, 40 nM, or 200 nM mithramycin) for 48 hours.    From Wordnik.com.  [PLoS ONE Alerts: New Articles] Reference

C) Freshly isolated human monocytes were cultured overnight in 5\% FBS and subsequently starved for 2 hours followed by pre-treatment with either methanol (vehicle control) or mithramycin (200 nM) in minimal media.    From Wordnik.com.  [PLoS ONE Alerts: New Articles] Reference